The concentration of uric acid in the blood is too high to be completely dissolved (supersaturated), forming sodium urate crystals to precipitate in soft tissues, causing gout attacks. Reducing the concentration of uric acid in the blood is the key to the prevention and treatment of gout. The basic treatment is to inhibit the production of uric acid (such as allopurinol, febuxostat, etc.) and promote the excretion of uric acid by the kidney (such as benzbromarone, probenecid, etc.) . During the onset of pain, anti-inflammatory and analgesic drugs, such as colchicine, are also needed.

　　Diet is closely related to the production of uric acid

　　Although there is no uric acid in food (this is because most animals have very little uric acid in their blood or tissues, which is very different from humans. It is even more important in plants. No uric acid), but a substance called "purine" (also known as "purine") in food is almost everywhere, and it is metabolized into uric acid in the human body. In fact, uric acid in human blood is indirectly derived from purine, so it is not difficult to understand that people with gout and high uric acid need to control the content of purine in the diet, which is called the "low purine diet."

　　The textbook says that during the acute attack of gout, a low-purine diet refers to the purine content of the daily diet