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How to diagnose submandibular gland inflammation

BY Berton Gladstone 2019-12-04

  The retrograde inflammation of the submandibular gland due to obstruction and stenosis of the catheter, called submandibular glanditis, is often complicated by salivary stones. The clinical manifestations are mainly submandibular gland enlargement, pain, and purulent discharge from the catheter orifice. The incidence of adults with this disease is high and most of them are chronic manifestations. At present, there are more effective therapeutic drugs and methods, and the cure rate is higher. Those who do not receive treatment in time and those who do not receive regular treatment have poor efficacy, so early treatment is the key.

  The submandibular gland is a salivary organ, Located under the jaw. There are two types of inflammation. One is viral infection, mainly local swelling, pain, fever, and white blood cell decline. One is caused by bacteria, that is, the submandibular gland duct in the mouth is blocked to cause swelling of the submandibular gland, and some people have repeated attacks for a long time. Bacterial infections require anti-inflammatory treatment, pay attention to oral hygiene. So, how to diagnose submandibular gland inflammation?
   Check items: CT contrast scan, hemogram, superficial organ color Doppler ultrasound, blood biochemistry, peripheral hemogram, white blood cell count are mostly normal or slightly increased, and lymphocytes are relatively increased. When there are complications, the white blood cell count can be increased, and occasionally leukemia-like reactions. Serum and urine amylase determination, 90% of patients with mild and moderate increase in serum amylase, help diagnosis. The degree of amylase increase is often proportional to the degree of submandibular gland swelling. However, its increase may also be related to pancreatic and intestinal serous adenopathy.
   Serological examination, neutralizing antibody test: low titers such as 1:2 indicate current infection. In recent years, the application of gel hemolysis test is basically consistent with the neutralization test, and the detection of neutralizing antibody is simpler and faster, but the method needs further improvement. Complement fixation test: It has the value of auxiliary diagnosis for suspicious cases. The titers of the double serum (early course and 2 to 3 weeks) have more than 4-fold increase, or the serum titer of 1:64 has diagnostic significance. If conditions permit, it is advisable to measure both S and V antibodies. An increase in S antibody indicates a recent infection. An increase in V antibody and an increase in S antibody only indicate that they have been infected in the past.
   Hemagglutination inhibition test: The virus-infected chicken embryo, its amniotic fluid and allantoic fluid can agglutinate the erythrocytes of the chicken. The convalescent serum of patients with submandibular gland inflammation has a strong inhibitory effect on agglutination, while the inhibitory effect of early serum is Weak, if the difference between the two determinations is more than 4 times, it is positive.
  Routine examination of urine, proteinuria, red and white blood cells, etc. may appear in the urine when the kidney is involved, and even urine changes similar to nephritis. Electrocardiogram examination, electrocardiogram when combined with myocarditis: arrhythmia, low T wave, ST segment depression.

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